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dc.contributor.authorTerzi M.
dc.contributor.authorDemir O.
dc.contributor.authorOnar M.
dc.date.accessioned2020-06-21T09:37:04Z
dc.date.available2020-06-21T09:37:04Z
dc.date.issued2011
dc.identifier.issn1309-0720
dc.identifier.urihttps://doi.org/10.4328/JCAM.295
dc.identifier.urihttps://hdl.handle.net/20.500.12712/4657
dc.description.abstractSystemic lupus erythematosus (SLE) is a complex multisystem disease that may involve the central and peripheral nervous systems. Common clinical findings include seizures, depression, psychosis, aseptic meningitis, headache, cranial and peripheral neuropathies. The incidence of stroke as a neurological involvement in patients with SLE is between 3-20%. Possible reasons for the development of stroke are suggested as hypercoagulability and thrombosis due to antiphospholipid antibodies, cerebral vasculitis, cerebral embolus due to Libman Sacks endocarditis and hypertension due to the disease itself or long term corticosteroid use. We present clinical, laboratory and radiological imaging findings that confirm cerebral vasculitis of a SLE case with the clinical presentation of acute stroke in this study.en_US
dc.language.isoengen_US
dc.publisherDerman Medical Publishingen_US
dc.relation.isversionof10.4328/JCAM.295en_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectAphasiaen_US
dc.subjectLupusen_US
dc.subjectStrokeen_US
dc.subjectVasculiten_US
dc.titlePure motor aphasia developed after cerebral vasculitis due to systemic lupus erythematosusen_US
dc.title.alternativeSistemik lupus Eritematozusa bağlı serebral vaskülit sonrası gelişen pür motor afazien_US
dc.typearticleen_US
dc.contributor.departmentOMÜen_US
dc.identifier.volume2en_US
dc.identifier.issue3en_US
dc.identifier.startpage116en_US
dc.identifier.endpage118en_US
dc.relation.journalJournal of Clinical and Analytical Medicineen_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US


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